SHL can affect different people very differently. SHL is usually unilateral (that is, it affects only one ear); and is often accompanied by tinnitus. Sudden hearing loss is associated with vertigo in between 20 to 60 of patients (Rambold et al, 2005). Orthostatic tinnitus is also possible. Many vascular and hematologic pathologies have been associated with SSNHL. The disease processes can occur at any level along this part of the ear. Sensorineural hearing loss – this refers to problems occurring in the cochlea (the most common site of disease), cochlear nerve or brain stem, resulting in abnormal or absent neurosensory impulses. Prognosis – recovery is generally good but chronic otitis media may lead to middle-ear effusion or tympanic membrane perforation. Presentation – there is a sudden, unilateral hearing loss associated with vertigo and tinnitus.
70 of Sudden sensorineural hearing loss patients also suffer from tinnitus. Incidents of Sudden sensorineural hearing loss are mostly unilateral, only 2 experience a case of bilateral sudden sensorineural hearing loss. The sequelae of a sudden loss of hearing can be significant. There are a number of causes of SOHL, both conductive and sensorineural (Table 1). Excessive straining might cause a perilymph fistula, however, tinnitus and vertigo would also usually be present. Sudden senorineural hearing loss (SSNHL) has wide age distribution, but occurs most commonly in people aged 50-60 years, usually affects one ear only, and can be associated with tinnitus and vertigo. Sudden senorineural hearing loss (SSNHL) has wide age distribution, but occurs most commonly in people aged 50-60 years, usually affects one ear only, and can be associated with tinnitus and vertigo. SSNHL may be the presenting symptom of M ni re’s disease a clinical diagnosis, which classically presents with sudden unilateral hearing loss, tinnitus, and vertigo.
SNHL is generally permanent and can be mild, moderate, severe, profound, or total. The most common kind of sensorineural hearing loss is age-related (presbycusis), followed by noise-induced hearing loss(NIHL). M ni re’s disease is characterized by sudden attacks of vertigo, lasting minutes to hours preceded by tinnitus, aural fullness, and fluctuating hearing loss. Sound localizes to affected ear (ear with conductive loss) in unilateral cases. Sudden sensorineural hearing loss is usually unilateral and can be associated with tinnitus and vertigo. In most cases the cause is not identified, although various infective, vascular, and immune. Idiopathic sudden sensorineural hearing loss (SSNHL), also known as sudden deafness, is defined as the onset of unexplained one-sided SNHL in less than 72 hours. All reports describing treatments indicated that earlier initiation of treatment was associated with better hearing outcome. Many patients, especially those with milder hearing loss, can experience some degree of spontaneous improvement, though rarely to normal. At one extreme is sudden onset of pure vertigo with no auditory symptoms.
Sudden Hearing Loss
Sudden sensorineural hearing loss (SSNHL) poses a diagnostic and therapeutic dilemma. The hearing loss is variable in severity and is usually unilateral, although bilateral involvement up to 4 has been reported. The differential diagnosis in patients with SSNHL is broad and could be divided into neoplastic, vascular, traumatic, infectious, metabolic, neurologic, immunologic, toxic, cochlear, and many other causes. Patients usually complain of associated aural fullness, tinnitus, and possible vertigo. Hearing loss can be categorized as conductive, sensorineural, or both. Leading causes of conductive hearing loss include cerumen impaction, otitis media, and otosclerosis. Otosclerosis typically presents as progressive bilateral conductive hearing loss in middle-aged white women. Postnatal infection associated with sensorineural hearing loss (e.g., meningitis). Sudden, fluctuating, unilateral hearing loss, tinnitus, episodic vertigo. The symptoms include hearing loss, tinnitus, vertigo, imbalance, pressure, and facial weakness and numbness. A sudden decrease in hearing, often associated with viral infection or vascular occlusion, occurs in about one in eight AN patients. The dura has sensory fibers that can transmit the sensation of the pressure. The headache that results from the acoustic neuroma can be dull or aching in quality and is usually unilateral. The tinnitus was associated with sudden right-sided hearing loss and vertigo, which occurred about 18 months before. The decrease of afferent input caused by SSHL could induce tinnitus connected to central neural changes. It also affects hearing, with tinnitus (usually a buzz or hum) and hearing loss (usually of low tones). The hearing loss can be detected even between attacks of vertigo. With acute unilateral hemispheric depression, such as caused by a middle cerebral artery occlusion, the fast component to the opposite side is depressed. B) Quinine like aspirin can cause reversible SNHL associated with tinnitus and can occur in both healthy and malaria patients. Sudden SNHL (high frequency) within 48-72 hours of commencing OKT3 has been reported followed by complete resolution within 2 weeks after discontinuation of the drug. 31 Two weeks later there was complete resolution of the deafness and the associated tinnitus and dizziness. The hearing loss is either unilateral or bilateral, low frequency and usually reverses spontaneously although there are a few documented cases where the SNHL has not reversed.
Sensorineural Hearing Loss
A sudden hearing loss occurs in about 25 of patients with acoustic neuroma. Tinnitus is very common in acoustic neuroma, and is usually unilateral and confined to the affected ear. Tinnitus is present in the ear to a variable degree, and the hearing loss sometimes is preceded by the onset of tinnitus. The more severe the loss, the lower the prognosis for recovery, and profound losses have an exceptionally poor prognosis. Herpes zoster oticus also can cause a sudden sensorineural hearing loss, although it is a clinical entity clearly distinct from idiopathic sudden sensorineural hearing loss. Additionally, mid- and high-frequency hearing loss are more commonly associated with AN than are low frequency losses, and electronystagmographic (ENG) abnormalities are more common with AN. Idiopathic sudden sensorineural hearing loss is a medical emergency that requires urgent diagnosis and treatment, and the adaptation of a proper protocol for management is a priority. The aim of this study was to determine and identify as early as possible those factors that play the important role in the prognosis of the condition, to describe the experience, and to suggest a treatment protocol that can be adopted in a tertiary hospital, such as Hamad General Hospital. ISSNHL is usually unilateral and is often accompanied by tinnitus, vertigo, or both. However, it can also refer to such feelings as lightheadedness, unsteadiness, confusion, giddiness, or nausea. The attacks are usually associated with tinnitus and/or aural fullness. Vestibular Neuronitis is characterized by the sudden onset of disabling vertigo often associated with nausea and vomiting without auditory symptoms. There are no definitive symptoms that will clearly point to a diagnosis of PLF, however, symptoms may be similar to those of Meniere’s disease or endolymphatic hydrops.49-50 Typically, a fluctuating unilateral sensorineural hearing loss occurs.
Sudden sensorineural hearing loss (SSNHL) involves an acute unexplained hearing loss, nearly always unilateral, that occurs over less than a 72-hour period. SSNHL can occur at any age, but most commonly affects patients 43 to 53 years of age 4. Some studies have suggested that diet (those low in fresh vegetables), low folate levels, and metabolic syndrome are associated with an increased risk of SSNHL 5-7. Int Tinnitus J 2002; 8:127.